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Based on gel filtration analysis and site-directed mutagenesis, it was suggested that a disulfide bond joined two monomers at least in AtAPR1 (Kopriva and Koprivova, 2004)

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Therefore, future research should include more human-based clinical trials that account for individual physiological and disease-related variations, tailoring dietary interventions and treatment strategies to the specific microbiome and metabolic responses of each patient