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The immunohistochemical protocol for GPX1 applied in this study was slightly modified from our previous procedures for interleukin-1 and inducible nitric oxide synthase (Reddy et al
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Xu's study [60] found that Pio, a peroxisome proliferatoractivated receptor agonist, could induce apoptosis and compensate for this effect by activating autophagy in TSPCs, thereby improving TSPC dysfunction
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