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Lipid droplets and peroxisomes are co-regulated to drive lifespan extension in response to mono-unsaturated fatty acids

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177 During T cell activation, the TCR complex aggregates with signaling molecules in lipid rafts, such as the Src family lymphocyte-specific protein tyrosine kinase (Lck), thereby increasing signal transduction (Fig

TEAD inhibitors (estrogen receptor, ER) significantly altered metabolic pathways, such as AMPK and PI3K/AKT signaling and reduced the expression of lipid metabolism related genes such as SREBP1 and 3-hydroxy-3-methylglutaryl-Coenzyme A reductase (HMGCR) (responsible for controlling cholesterol synthesis) by directly binding to their promoters to alleviate hepatic steatosis in HFD-induced MASLD male mice [13]