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These peripheral patterns support a testable hypothesis that toxic metal burden, redox-sensitive inflammatory signaling, and prostaglandin-pathway activation converge in biomarker-defined ASD subgroups

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*Correspondence: David Wasilewski, [email protected] Krasemann, [email protected] These authors have contributed equally to this work Present address: David Wasilewski, Department of Neurosurgery, CharitUniversittsmedizin Berlin, Corporate Member of Freie Universitt Berlin, Humboldt-Universitt zu Berlin, and Berlin Institute of Health, Berlin, Germany This article was submitted to Cellular Neuropathology, a section of the journal Frontiers in Cellular Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
