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Therapies like BPC-157 may one day become supportive tools in this toolkit, but they must be considered experimental
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292 In addition, intermittent hypoxia induces the sympathetic nervous system to increase vascular resistance, down-regulates expression of the kallikrein-kallistatin vasodilator pathway, and activates the renal renin-angiotensin-aldosterone system to cause vasoconstriction
The FDA-approved drug diflunisal can also inhibit ACMSD, providing a structural base from which derivatives can be designed 237
