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The mechanical activation of ENaC is, in part, predicated on N-glycosylation of asparagines, which are thought to act as anchoring points for extracellular tethers that interact with the extracellular matrix and/or the glycocalyx (16, 137)

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(9) When combined, these peptides appear to exert a synergistic action on the pituitary gland, which might result in enhanced growth hormone production

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NRF2 also interacts with oncogenic signaling pathways frequently deregulated in HCC, such as PI3K/AKT, mTOR, and NOTCH1