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doi: 10.1016/j.bbagen.2014.11.010 211 OsorioJSJiPDrackleyJKLuchiniDLoorJJ

A detailed mapping of disulfidptosis in nervous system disease may reveal the network regulating the crucial targets of the pathological process and provide new insights into potential clinical treatment directions for neurological disorders

This ATP-generating system is distinguished by its unique genomic architecture and serves as a critical regulatory node integrated with broader mitochondrial homeostatic networks

doi: 10.1007/s40257-014-0107-3

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Multiple binding partners like Beclin-1-UVRAG [61, 62], ATG14L/Barkor [63, 64], and AMBRA1 [65] positively regulate Beclin-1 in autophagy, while BCL2 and BCLxL from the anti-apoptotic BCL2 family inhibit autophagy