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Furthermore, inhibiting TLR4 can also modulate gut microbiota homeostasis and the MyD88/NF-B axis in ulcerative colitis (164), indicating that TLR4 acts not only as a key sensor of innate immunity but may also regulate neuroimmune crosstalk and gut microenvironment homeostasis
E.HarmonS
Nevertheless, they are still useful as second-line agents to induce remission in CD when other agents, such as biologics fail[1]
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Al-Tayyib, A., Koester, S., Langegger, S
