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Indeed, niacin activates myeloid cells derived from GBM patients in vitro, leading to their enhanced release of cytokines such as TNF and IL-6, and niacin-treated monocytes attenuate the growth of GBM patient-derived BTICs [11]


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Layer thickness and cell counts were analysed with ImageJ software

NaBPC157 was administered either intragastrically or intraperitoneally

Imbalance in intestinal microflora constitution could be involved in the pathogenesis of inflammatory bowel disease
