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Dysregulated Iron Metabolism and Ferroptosis Iron dyshomeostasis is a wellestablished contributor to dopaminergic neurodegeneration in PD

Colloids, by contrast, remain intravascular, so their volume-expanding effect can be greater (roughly a 1:1 effect with 25% albumin) but with risk of reactions

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Moreover, the use of CB-839 in combination with ABT-199 (BCL-2 inhibitor) facilitated mitochondrial depolarization, decreased cellular oxidative phosphorylation capacity, and induced apoptosis in OCI-AML2 and MORM-14 cell lines [20]