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Pharmacologic inhibition of CCR9-mediated trafficking of these cells reduced the severity of arthritis in animal models, suggesting that recirculation of arthritogenic immune cells between the gut and the joints may contribute to the pathogenesis of RA [3]

With Kropski and several VUMC colleagues, Banovich and Blackwell lead a major research initiative to uncover the molecular drivers of pulmonary disease

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Cell culture ZH-161, ZH-305 and T-269 GIC lines were isolated from surgically removed glioblastomas 34,35

One is by up-regulating a signaling protein called VEGF (vascular endothelial growth factor), which encourages the growth of new capillaries