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While GLP-1 therapies work through incretin pathways, cagrilintide activates amylin receptors located in key metabolic control centers of the brain[2]

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Physiol Rev (2011) 91(3):1071121

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Furthermore, DEFA1 is only one candidate out of numerous other potentially functional -defensins in the intestine of the horse, but nothing is known about their antimicrobial properties [41]

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