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Consequently, LDL builds up in the circulation, because there are insufficient receptors to bind the LDL and transfer the cholesterol into the cells

Reverse metabolomics for the discovery of chemical structures from humans

4.1.3 Other metabolic types In breast cancer cells under nicotine exposure, CEBPB upregulates GFAT to boost HBP activity, triggering elevated O-GlcNAcylation that disrupts CHOP heterodimerization, which in turn amplifies CEBPBs transcriptional activation of EMT drivers and metastatic pathways via a self-sustaining signaling circuit [14]