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In contrast, hepatic copper deficiency mainly promotes hepatic lipid synthesis[12]

Blockage of C-X-C motif chemokine receptor 2 (CXCR2) suppressed uric acid (UA)-induced cardiac remodeling

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This was associated with substantive mobility of the RAMP2 linker and TM helix, which had weaker interaction with the CTR TM bundle compared to RAMP1 and RAMP3 in AMY 1 R and AMY 3 R complexes

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It appears that the redox imbalance is an important etiologic contributor to insulin resistance, beta-cell dysfunction, and impaired glucose tolerance, all resulting in the development of type 2 diabetes (Wright et al