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The in-depth analyses of the results revealed that the proteins enolase, neuropolypeptide h3, ATP synthase, and fructose bis-aldolase (FBA) were identified as oxidized in brain from subjects at each stage of AD, late-stage AD, EAD, and MCI, consistent with the concept that their oxidation is an early event in and may contribute to progression of AD

Inhibition of miRNA binding improved cardiac function and restored mitochondrial fatty acid oxidation in animal models, thus highlighting a novel potential therapy for heart failure (36)

Eur J Neurosci 22(9):21992206 Shimamura M, Zhou P, Casolla B, Qian L, Capone C, Kurinami H, ladecola C, Anrather J (2013) Prostaglandin E2 type 1 receptors contribute to neuronal apoptosis after transient forebrain ischemia

Graphene matrix for signal enhancement in ambient plasma assisted laser desorption ionization mass spectrometry