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Further, C3 and C4 were negatively correlated with SLEDAI 2000 ( = 0.544 and 0.418) and with SLE-DAS ( = 0.290 and 0.242)

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Together, these results indicate that decreased GSH/GSSG redox/antioxidant capacity and increased oxidative stress in the autism brain may have functional consequence in terms of a chronic inflammatory response, increased mitochondrial superoxide production, and oxidative protein and DNA damage.