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doi:10.1016/j.cplett.2018.08.022 160

Medicine (Baltimore) 102:e36759

Hence, although these sleep deprivation-induced immune-mediated mechanisms in cancer warrant further confirmation in humans, the importance of the immune function in the anti-tumor host defense is well recognized 131 , thus suggesting that the impaired immune response after sleep deprivation may represent a plausible mediator of the associated increased risk for cancer as described in animal models and in humans

doi: 10.1056/NEJMoa2001191

Growth failure with normal serum RIA-GH and low somatomedin activity: somatomedin restoration and growth acceleration after exogenous GH