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In particular, MPTP intoxication typically downregulates key autophagy regulators (LC3-II, beclin-1, parkin, PINK1), but schisandrin A treatment partially preserved the levels of these proteins, implying that it enhances autophagic clearance of toxic aggregates (59, 60)

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This cellular impairment of EBV control can be observed by increased antibody responses to EBV and increased reactivation in patients with LC [8, 128,129,130, 133,134,135,136,137,138,139,140]

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In addition, many mechanistic questions remain regarding the discrete synapses and circuits that are impacted by presynaptic GPCRs to modify drug intake and seeking