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The Nrf2regulated Gpx and Trx are verified to suppress inflammatory response.54, 55 Cyclooxygenase2 (COX2), inducible nitric oxide synthase (iNOS), and tumor necrosis factor (TNF) are significantly elevated in the Nrf2deficient mice, indicating an inhibitory function of Nrf2 toward proinflammatory mediators.56 Additionally, the overexpression of HO1, a Nrf2 downstream target, attenuates TNFinduced oxidative stress and interleukin (IL)3 via the suppression of the DNAbinding activity of activator protein1 (AP1).57 Similarly, Nrf2dependent induction of NQO1 downregulates lipopolysaccharide (LPS)induced expression of TNF and IL1, thereby impairs the inflammatory response.58 To conclude, another significant role of Nrf2 is to suppress inflammatory response and protect cells against inflammatory injury and inflammationinduced carcinogenesis

This disruption results in an imbalance in adipokine secretion: levels of the insulin-sensitizing and anti-inflammatory factor adiponectin decrease significantly, while pro-inflammatory factors such as leptin, TNF - and MCP-1 increase

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Copper and zinc interact significantly in their joint toxicity to Chlamydomonas reinhardtii : insights from physiological and transcriptomic investigations