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These findings confirmed the role of altered glutamatergic and GABAergic neurotransmission in the pathophysiology of depression, as described in a previous review [36]
LiuXXiXJiaAZhangMCuiTBaiXet al
Instead, AA147-dependent protection against glutamate-induced oxidative toxicity is primarily mediated through compound-dependent activation of the NRF2 oxidative stress response
doi:10.3390/ijms19071987 Campbell JD, McDonough JE, Zeskind JE, et al
Targeting FLT3 Mutations in AML: Review of Current Knowledge and Evidence
Ann Plast Surg (2020) 84(5S Suppl 3):S20814
