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While genetic variation in TSD susceptibility has been reported in reptiles and amphibians (e.g., laboratory studies have shown genetic variation in specific populations), whether this variation provides sufficient adaptive capacity remains an open question (Urban et al., 2013)

Targeting the NLRP3 inflammasome in inflammatory diseases

A possible mechanism by which LA regulates rearrangement of actin filaments to promote tight junction reassembly is through the myosin light chain (MLC) pathway

Personalizing the treatment program for the individual patient also has to factor in whether additional therapies connected to the radiation, such as the use of hormone therapy or other systemic treatments, are needed to help maximize the ability to get rid of the disease

Supports transition from preclinical to clinical IBD research Favorable safety profile across all preclinical toxicology studies Effective in TNBS-colitis and cysteamine-duodenal ulcer models