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Increased levels of programmed cell death 5 (PDCD5) subsequently ameliorated progressive fibrosis and cardiac dysfunction by inhibiting histone deacetylase 3 (HDAC3), suggesting that PDCD5 serves as an endogenous negative feedback regulator in progressive cardiac fibrosis [109]
Sinha, R., & Kohaar, I
Omega-3 fatty acids support anti-inflammatory effects
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The underlying molecular mechanisms, like mitochondrial dysfunction, oxidative stress, and immune-mediated responses, are discussed in detail
Importantly, these effects may persist long after cessation of steroid use, as highlighted in both clinical and experimental studies
