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NAD + regeneration rescues lifespan, but not ataxia, in a mouse model of brain mitochondrial complex I dysfunction

Twenty-four-hour supplementation of aged neurons with the NAD precursor nicotinamide and the Nrf2 redox modulator EGCG restored GTP levels to youthful levels and mobilized endocytosis and lysosomal consumption for autophagy via the respective GTPases Rab7 and Arl8b

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36 Results showed that slowly migrating higher molecular weight protein complexes (e.g., SNAP-25, N -ethylmaleimide sensitive factor) occurred only at relatively high (25 mM) acrolein concentrations

Modified-release pulmonary delivery systems for labile bioactives: Design, development, and applications