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References Liepinsh, E., Kalvinsh, I

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The possible mechanisms that account for the beneficial response seen with L-carnitine in AMI are probably multifactorial and include, in part, an improvement in myocardial mitochondrial energy metabolism in the heart due to its ability to enhance the transport of long-chain fatty acids from the cytosol to the mitochondrial matrix where -oxidation occurs, removing toxic fatty acid intermediates, lessening ischemia secondary to elevated long-chain fatty acid concentrations, and replenishing the depleted carnitine concentrations seen in ischemic, infarct, and failing myocardium

Figure 5 MSA-DH activity experiments in the presence of the cofactor NAD + or NADP + (2 mM) revealed closely related enzyme activities (Figure 5B)

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