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Description
Direct scavenging of free radicals (especially ROS), inhibition of ROS manufacturing enzymes, and preservation of mitochondrial integrity and function affect redox signaling by activating nuclear factor erythroid-derived 2-like 2 (Nrf2) and peroxisome proliferator-activated receptor alpha (PPAR), and the synthesis of heat shock proteins (HSPs), thioredoxins, sirtuins, and other antioxidant molecules are pathways through which L-carnitine and its derivatives modulate oxidative stress conditions [14, 38]
Jiang X, Stockwell BR, Conrad M

Cerebral perfusion changes after Acetyl-L-Carnitine treatment in early Alzheimers Disease using single photon emission computed tomography

L-Carnitine Appie cider inzent 1 1

Conclusions This meta-analysis has shown that L-carnitine supplementation significantly reduces FPG, insulin, HOMA-IR, and HbA1c levels

Our results suggest that some individuals with ARTHS might benefit from ALCAR treatment
