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These include: (i) the polyol pathway, enhanced formation of advanced glycation end-products (AGEs)

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For example, deletion of hepatic Hmgcs2 using antisense oligonucleotides resulted in a significant increase in hepatic neutral lipid accumulation following regular chow and high-fat feeding conditions in both neonates and adult mice ( hmgcs2 results in a massive deposition of hepatic triglycerides in neonates ( Srebp1 , Chrebp1 , and Scd2 (Fabbrini et al., 2008