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VPA is extensively metabolized by the liver via glucuronic acid conjugation, mitochondrial and cytosolic (endoplasmic reticulum) -oxidation to produce multiple metabolites, some of which may be biologically active (Fig

A primary mechanism underlying this toxicity is oxidative stress, evidenced by increased lipid peroxidation (malondialdehyde MDA) and depletion of antioxidant defenses (superoxide dismutase SOD) within reproductive tissues, leading to inflammation and cellular damage 7

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