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Overexpression of the constitutive active hEro1L mutant showed a slight oxidation of the ER redox state both in control (DMSO treated) and in Epx treated condition, although it did not recover the ER reductive shift following proteasome inhibition at all (Fig

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RSL3, in combination with cisplatin, increased lipid peroxidation and decreased tumor growth in the preclinical model (Yuan et al

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Increased GSH levels and GCL expression occur in many tumors and have been implicated to confer drug and/or radiation resistance to tumors of ovarian, lung, prostate and colorectal origins (Lu, 1999a)