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Hyperhomocysteinemia also determines Nitric Oxide (NO) metabolism impairment in endothelial cells (including Nitric Oxide Synthase expression, localization, activation, and activity) leading, together with ROS-induced local microinflammation, to endothelial dysfunction [39]

Significantly, tumor growth was delayed in the KBiF 4 @HSA + X group

& Pugliese, A

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Considering this and the evolutionary divergence of Nematoda, we hypothesise that diversification of CAT within metazoans is relatively recent compared to the long evolutionarily history of this enzyme

At this point, medical treatment is essential