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Mol Biol Rep 2014;41:2851-7

Neuroinflammation modulation: Post-TBI microglial activation shifting toward the M1 pro-inflammatory phenotype (elevated TNF-, IL-1, IL-6, iNOS) amplifies secondary neuronal damage

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Recently, also, the modification of the redox state of cysteine residues of certain proteins, which is a widespread mechanism in the regulation of protein function, has been proposed to be involved in signaling pathways

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