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doi:10.1016/j.clnesp.2017.01.002

NO generated by iNOS could interact with lipid peroxides and secondary lipid radical intermediates catalyzed by 15-LOX (70) and substitute GPX4 as an anti-ferroptotic defense, which renders M1 macrophages with high resistance to pharmacologically induced ferroptosis

The rest of the atoms within C1-27 interact with the H-site causing rearrangement of several side chains and a 1 shift in the 4b helix (residues 122132) with respect to the GSTO1-GSH structure (1EEM) (Fig

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