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doi: 10.1093/nar/gkaa1074

ERp57 chaperon protein protects neuronal cells from A-induced toxicity

As described herein, conditions resulting from ischemia, and injuries caused by ischemia or ischemia-reperfusion, can induce apoptotic cell death in an affected cell, tissue, and/or organ, leading to damage and dysfunction

178 This approach aims to streamline treatment adjustments and enhance long-term outcomes

490 The ability of iron chelators or ferroptosis inhibitors to reverse cardiac damage during both acute and chronic I/R injury highlights ferroptosis as a promising new treatment strategy for myocardial I/R injury

doi: 10.1002/dev.21801