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Nuclear exclusion of p53 reportedly leads to translocation of p53 to mitochondria and transcription-independent induction of apoptosis [28], which may explain the therapeutic effect of FOXO4-DRI

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This advantage is especially pronounced when the antibodies are directly generated with a SpyTag, such as we have established for Pioneer and HuCAL antibody libraries 37,38 , but it is easy to reclone any human antibody fragment for SpyTagged expression in E

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