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For these reasons, SKM is a very attractive target for the development of novel classes of antidiabetic drugs

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2021 Feb 1;22(2):217-218

Although neurons activate compensatory NAD + salvage through nicotinamide phosphoribosyltransferase (NAMPT) [108], this pathway fails to offset PARP-1-driven NAD + catabolism during stroke [106], resulting in sustained sirtuin suppression and disrupted mitochondrial dynamics

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and (OTKA K131458) to P.C., from the Semmelweis University (STIA_18_M/6800313263, STIA-KFI-2020/132257/AOMBT/2020) and from the Department of Molecular Biology of the Semmelweis University (Baron Munchausen Program 2023/1) to C.S