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In the original cytoprotection studies, pancreatitis was known as a particular target for the standard cytoprotective agent activity [81], combined with the stomach and duodenal lesions [29,30]

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The peptide does not suppress natural hormone production, cause organ toxicity at therapeutic doses, or produce withdrawal effects upon discontinuation

In vivo c-Met pathway inhibition depletes human glioma xenografts of tumor-propagating stem-like cells

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