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GSH is highly resistant to intracellular degradation
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Suppression of the murine osteoblasts MC3T3-E1 proliferation by dexamethasone (Dex) was associated with G1 phase delay and apoptosis induction accompanied by p53-dependent activation of p21 and the up-regulation of proapoptotic genes NOXA and PUMA [24]
A considered increase was recorded in the renal antioxidant SOD in silymarin standard, NSO, and NSS groups
These targets include ACC2 and diacylglycerol acyltransferase 2 (DGAT2)
