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n = 6 mice/group, mean SD were presented

1 To date, multiple mechanisms of tissue damage have been implicated in SLE, including oxidative stress and free radicals, fibrosis, complement-mediated pathways, and a panoply of enzymatic cascades including matrix metalloproteinases, the renin-angiotensin system, plasmin, and kallikreins

Interestingly, when compared to other leukocytes, such as macrophages and lymphocytes, neutrophils consume glutamine at the highest rates [98,99]

Other AD-associated pathology, including neuroinflammation and oxidative stress, may also impair the development of oligodendrocytes and myelination [111, 115]

pylori has been cured

Flow cytometry confirmed significant depletion of CD8 + T and NK cells in the spleens and peripheral blood of treated mice (Supplementary Figure S8)