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In preclinical cellular models, FOXO4-DRI has been reported to: Disrupt FOXO4p53 interaction and reduce nuclear co-localization in senescent cells Promote p53-dependent apoptosis preferentially in senescent, rather than proliferating, cells Decrease markers of cellular senescence (such as SA--gal positivity and selected SASP factors) in treated cultures In animal studies, administration of FOXO4-DRI has been associated with: Reduction of senescence markers in select tissues Improvements in certain functional readouts (such as aspects of physical performance or organ function) in aging or therapy-damaged models Modulation of inflammatory and tissue-remodeling profiles linked to senescent-cell burden These outcomes vary depending on model, dosing regimen, route and experimental design, and remain limited to exploratory preclinical work

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Long-Term ERK Inhibition in KRAS-Mutant Pancreatic Cancer Is Associated with MYC Degradation and Senescence-like Growth Suppression

Initially, on day 7, BPC 157 rats had only small edema and inflammatory infiltrate, the connective tissue in more appropriate structure with only a small scar at the 14 days that would not disrupt the orientation as well as continuity of the muscle-tendon junction

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Spugnini et al