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Eosinophil accumulation preceded the onset of chronic cutaneous inflammation caused by Leishmania mexicana infection

Lazzeri L, Vannuccini S, Orlandini C, Luisi S, Zupi E, Nappi RE, et al

Rabe, C., Steenkamp, J.A., Joubert, E., Burger, J.F.W., Ferreira, D

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Organon, Netherlands)

Mitochondrial dysfunction associated with A and tau pathology in AD includes decreased ATP production, mitophagy, biogenesis (decreased expression of peroxisome proliferator-activated receptor gamma coactivator 1-alpha, PGC-1), activity of components of the OXPHOS system and other enzymes (e.g., 17-hydroxysteroid dehydrogenase type 10, HSD-10), mitochondrial membrane potential ( m ), and import of mitochondrial proteins, imbalance of mitochondrial dynamics (upregulation of dynamin-like protein-1, DRP-1), impairment of intracellular Ca 2+ homeostasis, membrane damage, interaction with adenine nucleotide translocator 1 (ANT1) and voltage-dependent anion channel 1 (VDAC1), and increased ROS production, apoptosis (increased cytochrome c release), and mPTP opening [8,63,68,72,73,74] (Figure 2)