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The segmentation framework is highly flexible and can be trained to identify vascular amyloid deposits, fibrinogen leakage, ischemic lesions, or other biomarkers associated with cerebrovascular aging and neurovascular dysfunction

M., Monette, R., Durkin, J

7 Clinical note: These mechanisms are derived from preclinical models

Without consistency across trials, it becomes difficult to draw firm conclusions or develop universal guidelines for use [45, 46]

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In this blend they are already combined, and the pairing has a mechanistic rationale: NAD+ supplies the coenzyme, while 5-Amino-1MQ blocks the NNMT enzyme that would otherwise drain NAD+ precursors [4]