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Description
Gluckman, E

In our experimental model, treatment with DDS (40 mg/kg) decreased TEAC, SGH, SOD and CAT in both tissues

doi: 10.1007/s11033-025-10632-x Summary Keywords diabetes mellitus, oxidative stress, AGEs, testicular damage, spermatogenesis impairment Citation Zhang W, Tong L, Jin B and Sun D (2025) Diabetic testicular dysfunction and spermatogenesis impairment: mechanisms and therapeutic prospects

Based on previous findings, oxytocin could be a promising hormone therapy for the management of ASD

Estrogen-Related Receptor maintains pancreatic acinar cell function and identity by regulating cellular metabolism

