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Leveraging these insights, we develop a BNIP3 antagonist peptide (B-017) that disrupts interactions between BNIP3 and BCL-2 executioner proteins, preserving mitochondrial integrity

Well, at least for a few weeks

Although the final outcomes of these two processes are the same, if senescence begins, it ensures that the lesion is efficiently removed via immunosurveillance, whereas, if apoptosis begins, it destroys the damaged cells without initiating an immune response [195]

The same applies to airway hyperreactivity as a consequence of inflammation

This is a more physiological approach compared to directly injecting growth hormone

MM: Conceptualization, Writing review & editing, Supervision