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In another experiment, mice with cAtg3-KO-induced cardiac dysfunction were treated with 5-amino-1-MQ

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Concentrations of 1,8-diMQ and 5-amino-1MQ ranging from 10 nM to 200 or 600 M, respectively, did not inhibit DNMT1 or PRMT3 (Figures 4A and 4B)

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Uniquely, Lactobacillus relative abundance was dramatically higher in the 5A-1MQ-treated group switched from WD to LD compared to all other groups (Fig

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