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Since the physiological level of ROS plays an important role in normal kidney functioning, any changes in redox equilibrium in the renal cortex and medulla may have serious consequences

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Mitochondrial ROS-derived PTEN oxidation activates PI3K pathway for mTOR-induced myogenic autophagy

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Pathological interactions between the brain and the heart have key pathophysiological roles in myocardial ischaemia and dysfunction in patients with Takotsubo syndrome