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Mitochondrial dysfunction and its relationship with mTOR signaling and oxidative damage in autism spectrum disorders

Glabridin exhibited therapeutic potential in the treatment of DN through improving renal function by repressing not only ferroptosis but also VEGF/p-Akt/p-ERK1/2 axis activation [247]

(E)CsInTe 2 - R 3 m

doi: 10.1007/s13197-015-1746-y

279 The production of 9( Z ),12( Z )-17-HODE, 9( Z ),12( Z )-8,17-DiHODE, and 9( Z ),12( Z )-8,16-DiHODE in vitro from LA by an of as yet undetermined CYP has also been reported in the rice blast fungus M

The findings were a reduction of ATP production in the cell line together with disruption of mitochondrial DNA (mtDNA) but without an increase in reactive oxygen species (ROS) or activity of caspase-3 and caspase-7, which would be the case for apoptosis (Janik-Karpinsa et al., 2022)