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In the overfat, malnourished, and immune-compromised host, the viral lifecycle can be altered, complementing an already weakened immune response, lead to severe pathogenesis and prolonged viral shedding, and can permit the emergence of virulent minor variants (36, 38, 39)

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The inactivity of the lipid repair enzyme GPX4 may result in elevated levels of lipid-based ROS, hence triggering ferroptosis [56]

PMID: 20370930

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