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Compounds that can either stabilize NRF2 or inhibit KEAP1, thereby enhancing NRF2's antioxidative response, are under investigation for their potential neuroprotective effects against radiation-induced damage [41]

These two isoforms have considerable similarities in their expression patterns and functions, despite their evolution at independent gene loci

The ability of mitochondria-targeted antioxidants confers greater protection against oxidative damage has been attributed to their abilities to cross the phospholipid bilayer of mitochondria and thus eliminating ROS (Oyewole and Birch-Machin, 2015)

S8F), which was further confirmed by the knockdown of Stat6 in NCTC1469 cells (Fig

Both can disturb sleep quality

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