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Meanwhile, increased mitochondrial GLS expression enhances GLN catabolism by Myc oncogene inhibition of miR-23, which in turn targets GLS [23]

25 Apart from GlyR all targeted antigens are predominantly cytoplasmic and it remains to be determined whether they can transiently exhibit an extracellular domain during neurotransmission and exocytosis that may account for pathogenicity

10.1158/0008-5472.CAN-08-1321 5 BarbosaN
Cisplatin in cancer therapy: molecular mechanisms of action

In this process, tryptophan is catalyzed by TPH to form 5-hydroxytryptophan

3(c) and (d), Table 3)
