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The roles of the ECS regulating immune and cognitive functions also support its modulation as a potential novel therapeutic target in AD

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Similar content being viewed by others Background Cardiovasular disease (CVD) is the world's leading cause of death, far outstripping infectious disease mortality from malaria, HIV/AIDS, and tuberculosis [1]

274,275 Therapeutic strategies targeting oxidative stress and mitochondrial dysfunction include using antioxidants like alpha-lipoic acid (ALA), which improves neuropathic symptoms and nerve conduction velocities in patients with DPN

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