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Association of HLA antigens with the clinical course of sarcoidosis and familial disease

Oxidative Stress and DNA Damage A primary mechanism by which toxins exert carcinogenic effects is through oxidative stress [14]

A study using 176 human lymphoblastoid cell lines exposed to the imine NAPQI used a novel approach to identify risk factors for APAP DILI.69 The IC 50 for APAP toxicity in each cell line was examined in relation to the genome-wide genotype for the cell line

Combinative treatment of beta-elemene and cetuximab is sensitive to KRAS mutant colorectal cancer cells by inducing ferroptosis and inhibiting epithelial-mesenchymal transformation

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